Ascorbic acid reduces the genetic damage caused by miltefosine (hexadecylphosphocholine) in animals infected by Leishmania (Leishamnia) infantum without decreasing its antileishmanial activity

Author:

Castelo-Branco Patrícia Valéria,Alves Hugo José,Pontes Raissa Lacerda,Maciel-Silva Vera Lucia,Ferreira Pereira Silma Regina

Funder

Coordenação de Aperfeiçoamento de Pessoal de Nível Superior - Brasil

Fundação de Amparo à Pesquisa e ao Desenvolvimento Científico e Tecnológico do MaranhãoFundação de Amparo à Pesquisa e ao Desenvolvimento Científico e Tecnológico do Maranhão (FAPEMA)

Publisher

Elsevier BV

Subject

Pharmacology (medical),Infectious Diseases,Pharmacology,Parasitology

Reference49 articles.

1. Avanços no tratamento da leishmaniose tegumentar do novo mundo nos últimos dez anos: uma revisão sistemática da literatura;Almeida;An. Bras. Dermatol.,2011

2. Repair and genetic consequences of endogenous DNA base damage in mammalian cells;Barnes;Annu. Rev. Genet.,2004

3. Sodium antimony gluconate induces generation of reactive oxygen species and nitric oxide via phosphoinositide 3-kinase and mitogen-activated protein kinase activation in Leishmania donovani-infected macrophages;Basu;Anti. Age Chemo,2006

4. Antiproliferative and ultrastructural effects of phenethylamine derivatives on promastigotes and amastigotes of Leishmania (Leishmania) infantum chagasi;Brasil;Parasitol. Int.,2017

5. Soy isoflavones have antimutagenic activity on DNA damage induced by the antileishmanial Glucantime (meglumine antimoniate);Cantanhêde;Drug Chem. Toxicol.,2015

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