Hirudin inhibit the formation of NLRP3 inflammasome in cardiomyocytes via suppressing oxidative stress and activating mitophagy-Reference-Cited by-同舟云学术

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Hirudin inhibit the formation of NLRP3 inflammasome in cardiomyocytes via suppressing oxidative stress and activating mitophagy

Published:2024-01 Issue:1 Volume:10 Page:e23077
ISSN:2405-8440
Container-title:Heliyon
language:en
Short-container-title:Heliyon

Author:

Luo Gang,Chen Li,Chen Mingtai,Mao Linshen,Zeng Qihu,Zou Yuan,Xue Jinyi,Liu Ping,Wu Qibiao,Yang Sijin,Liu Mengnan^ORCID

Publisher

Elsevier BV

Subject

Multidisciplinary

Reference33 articles.

1. ERK: a key player in the pathophysiology of cardiac hypertrophy;Gallo;Int. J. Mol. Sci.,2019

2. Myocardial hypertrophy is prevented by farnesol through oxidative stress and ERK1/2 signaling pathways;Souza;Eur. J. Pharmacol.,2020

3. Ginsenoside Rg3 attenuates angiotensin II-induced myocardial hypertrophy through repressing NLRP3 inflammasome and oxidative stress via modulating SIRT1/NF-kappaB pathway;Ren;Int. Immunopharm.,2021

4. NLRP3 inflammasome in neurological diseases, from functions to therapies;Song;Front. Cell. Neurosci.,2017

5. Mesenchymal stromal cells inhibit NLRP3 inflammasome activation in a model of Coxsackievirus B3-induced inflammatory cardiomyopathy;Miteva;Sci. Rep.,2018

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