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tBHQ attenuates podocyte injury in diabetic nephropathy by inhibiting NADPH oxidase-derived ROS generation via the Nrf2/HO-1 signalling pathway

  • Published:2022-09 Issue:9 Volume:8 Page:e10515
  • ISSN:2405-8440
  • Container-title:Heliyon
  • language:en
  • Short-container-title:Heliyon

Author:

Liu Ting,Li Chang-yan,Chen Hao,Liu Juan,Zhong Li-li,Tang Ming-min,Wang Wen-bo,Huang Jin-ping,Jiang Xu-shun

Funder

Health and Family Planning Commission of Sichuan Province

Publisher

Elsevier BV

Subject

Multidisciplinary

Reference40 articles.

1. The role of the complement system in diabetic nephropathy;Flyvbjerg;Nat. Rev. Nephrol.,2017

2. Podocyte injury: the role of proteinuria, urinary plasminogen, and oxidative stress;Raij;Am. J. Physiol. Ren. Physiol.,2016

3. Research progress on mechanism of podocyte depletion in diabetic nephropathy;Dai;J. Diabetes Res.,2017

4. Diabetes and kidney disease: role of oxidative stress;Jha;Antioxidants Redox Signal.,2016

5. Role of NADPH oxidase in metabolic disease-related renal injury: an update;Wan;Oxid. Med. Cell. Longev.,2016
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