Hyperactivation of mTOR and AKT in a cardiac hypertrophy animal model of Friedreich ataxia-Reference-Cited by-同舟云学术

Hyperactivation of mTOR and AKT in a cardiac hypertrophy animal model of Friedreich ataxia

Published:2022-08 Issue:8 Volume:8 Page:e10371
ISSN:2405-8440
Container-title:Heliyon
language:en
Short-container-title:Heliyon

Author:

Tong Wing-Hang^ORCID,Ollivierre Hayden,Noguchi Audrey,Ghosh Manik C.,Springer Danielle A.,Rouault Tracey A.^ORCID

Funder

NIH

Eunice Kennedy Shriver National Institute of Child Health and Human Development

Publisher

Elsevier BV

Subject

Multidisciplinary

Reference74 articles.

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3. mTOR regulates cellular iron homeostasis through tristetraprolin;Bayeva;Cell Metabol.,2012

4. Correction of half the cardiomyocytes fully rescue Friedreich ataxia mitochondrial cardiomyopathy through cell-autonomous mechanisms;Belbellaa;Hum. Mol. Genet.,2019

5. mTORC1 induces purine synthesis through control of the mitochondrial tetrahydrofolate cycle;Ben-Sahra;Science,2016

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5. SESN2-Mediated AKT/GSK-3β/NRF2 Activation to Ameliorate Adriamycin Cardiotoxicity in High-Fat Diet–Induced Obese Mice;Antioxidants & Redox Signaling;2023-08-03