Transgenic overexpression of the SUR2A-55 splice variant in mouse heart reduces infract size and promotes protective mitochondrial function
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
Multidisciplinary
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5. Genetic Discovery of ATP-Sensitive K + Channels in Cardiovascular Diseases;Circulation: Arrhythmia and Electrophysiology;2019-05
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