Disrupted nuclear import of cell cycle proteins in Huntington's/PolyQ disease causes neurodevelopment defects in cellular and Drosophila model-Reference-Cited by-同舟云学术

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Disrupted nuclear import of cell cycle proteins in Huntington's/PolyQ disease causes neurodevelopment defects in cellular and Drosophila model

Published:2024-02 Issue:4 Volume:10 Page:e26393
ISSN:2405-8440
Container-title:Heliyon
language:en
Short-container-title:Heliyon

Author:

Dubey Sandeep Kumar,Lloyd Thomas E.,Tapadia Madhu G.^ORCID

Publisher

Elsevier BV

Reference47 articles.

1. Huntington's disease gene (IT15) is widely expressed in human and rat tissues;Li;Neuron,1993

2. Widespread expression of the human and rat Huntington's disease gene in brain and nonneural tissues;Strong;Nat. Genet.,1993

3. Cellular localization of the Huntington's disease protein and discrimination of the normal and mutated form;Trottier;Nat. Genet.,1995

4. Widespread expression of Huntington's disease gene (IT15) protein product;Sharp;Neuron,1995

5. The biological function of the Huntingtin protein and its relevance to Huntington's Disease pathology;Schulte;Curr. Trends Neurol.,2011

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Clinical and genetic association studies reveal within-individual variability as an integral part of Huntington disease;2024-05-20

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