ZNF521 promotes acute myeloid leukemogenesis by suppressing the expression and acetylation of SMC3
Author:
Funder
National Natural Science Foundation of China
Publisher
Elsevier BV
Reference27 articles.
1. Identification of Evi-3, a novel common site of retroviral integration in mouse AKXD B-cell lymphomas;Justice;J. Virol.,1994
2. ZNF521 enhances MLL-AF9-dependent hematopoietic stem cell transformation in acute myeloid leukemias by altering the gene expression landscape;Chiarella;Int. J. Mol. Sci.,2021
3. ZFP521 regulates murine hematopoietic stem cell function and facilitates MLL-AF9 leukemogenesis in mouse and human cells;Garrison;Blood,2017
4. Transcriptional regulation of the proto-oncogene Zfp521 by SPI1 (PU.1) and HOXC13;Yu;Genesis,2016
5. Zfp521 promotes B-cell viability and cyclin D1 gene expression in a B cell culture system;Al Dallal;Leuk. Res.,2016
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