Atrial fibroblast-derived exosomal miR-21 upregulate myocardial KCa3.1 via the PI3K-Akt pathway during rapid pacing-Reference-Cited by-同舟云学术

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Atrial fibroblast-derived exosomal miR-21 upregulate myocardial KCa3.1 via the PI3K-Akt pathway during rapid pacing

Published:2024-07 Issue:13 Volume:10 Page:e33059
ISSN:2405-8440
Container-title:Heliyon
language:en
Short-container-title:Heliyon

Author:

Yuntao Fu,Jinjun Liang,Hua Fen Liu,Huiyu Chen,Dishiwen Liu,Zhen Cao,Wang Youcheng,Wang Xuewen,Ke Yuanjia,Yanni Cheng,Kexin Guo,Zhibin Peng,Mei Yang,Zhao Qingyan^ORCID

Funder

National Natural Science Foundation of China

Natural Science Foundation of Hubei Province

Publisher

Elsevier BV

Reference38 articles.

1. The clinical profile and pathophysiology of atrial fibrillation: relationships among clinical features, epidemiology, and mechanisms;Andrade;Circ. Res.,2014

2. Inflammation and the pathogenesis of atrial fibrillation;Hu;Nat. Rev. Cardiol.,2015

3. Evaluating the atrial myopathy underlying atrial fibrillation: identifying the arrhythmogenic and thrombogenic substrate;Goldberger;Circulation,2015

4. Extracellular vesicles from epicardial fat facilitate atrial fibrillation;Shaihov-Teper;Circulation,2021

5. MicroRNA-21 contributes to myocardial disease by stimulating MAP kinase signalling in fibroblasts;Thum;Nature,2008

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