11β-Prostaglandin F2α, a bioactive metabolite catalyzed by AKR1C3, stimulates prostaglandin F receptor and induces slug expression in breast cancer-Reference-Cited by-同舟云学术

11β-Prostaglandin F2α, a bioactive metabolite catalyzed by AKR1C3, stimulates prostaglandin F receptor and induces slug expression in breast cancer

Published:2015-09 Issue: Volume:413 Page:236-247
ISSN:0303-7207
Container-title:Molecular and Cellular Endocrinology
language:en
Short-container-title:Molecular and Cellular Endocrinology

Author:

Yoda Tomomi,Kikuchi Kyoko,Miki Yasuhiro,Onodera Yoshiaki,Hata Shuko,Takagi Kiyoshi,Nakamura Yasuhiro,Hirakawa Hisashi,Ishida Takanori,Suzuki Takashi,Ohuchi Noriaki,Sasano Hironobu,McNamara Keely May

Publisher

Elsevier BV

Subject

Endocrinology,Molecular Biology,Biochemistry

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4. Aldo-keto reductase 1C3 expression in MCF-7 cells reveals roles in steroid hormone and prostaglandin metabolism that may explain its over-expression in breast cancer;Byrns;J. Steroid Biochem. Mol. Biol.,2010

5. Type 5 17beta-hydroxysteroid dehydrogenase/prostaglandin F synthase (AKR1C3): role in breast cancer and inhibition by non-steroidal anti-inflammatory drug analogs;Byrns;Chem. Biol. Interact.,2009

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