Insights into how phosphorylation of estrogen receptor at serine 305 modulates tamoxifen activity in breast cancer
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
Endocrinology,Molecular Biology,Biochemistry
Reference38 articles.
1. Endocrine-responsive breast cancer and strategies for combating resistance;Ali;Nat. Rev. Canc.,2002
2. Estrogen receptor activation at serine 305 is sufficient to upregulate cyclin D1 in breast cancer cells;Balasenthil;FEBS Lett.,2004
3. Expression of the K303R estrogen receptor-alpha breast cancer mutation induces resistance to an aromatase inhibitor via addiction to the PI3K/Akt kinase pathway;Barone;Cancer Res.,2009
4. Phosphorylation of the mutant K303R estrogen receptor alpha at serine 305 affects aromatase inhibitor sensitivity;Barone;Oncogene,2010
5. Inflammation: an instigator of more aggressive estrogen receptor (ER) positive breast cancers;Baumgarten;Mol. Endocrinol.,2012
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