Pubertal FGF21 deficit is central in the metabolic pathophysiology of an ovine model of polycystic ovary syndrome-Reference-Cited by-同舟云学术

Pubertal FGF21 deficit is central in the metabolic pathophysiology of an ovine model of polycystic ovary syndrome

Published:2021-04 Issue: Volume:525 Page:111196
ISSN:0303-7207
Container-title:Molecular and Cellular Endocrinology
language:en
Short-container-title:Molecular and Cellular Endocrinology

Author:

Siemienowicz Katarzyna J.^ORCID,Furmanska Klaudia,Filis Panagiotis^ORCID,Talia Chiara,Thomas Jennifer^ORCID,Fowler Paul A.,Rae Mick T.,Duncan W. Colin

Funder

Medical Research Council

Publisher

Elsevier BV

Subject

Endocrinology,Molecular Biology,Biochemistry

Reference83 articles.

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4. Adverse metabolic phenotype of adolescent girls with non-alcoholic fatty liver disease plus polycystic ovary syndrome compared with other girls and boys;Ayonrinde;J. Gastroenterol. Hepatol.,2016

5. Fibroblast growth factor 21-deficient mice demonstrate impaired adaptation to ketosis;Badman;Endocrinology,2009

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