Altered FOXO1 activation in the programming of cardiovascular alterations by maternal diabetes-Reference-Cited by-同舟云学术

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Altered FOXO1 activation in the programming of cardiovascular alterations by maternal diabetes

Published:2019-01 Issue: Volume:479 Page:78-86
ISSN:0303-7207
Container-title:Molecular and Cellular Endocrinology
language:en
Short-container-title:Molecular and Cellular Endocrinology

Author:

Musikant Daniel,Sato Hugo,Capobianco Evangelina,White Verónica,Jawerbaum Alicia,Higa Romina

Funder

Agencia de Promoción Científica y Tecnológica of Argentina

Publisher

Elsevier BV

Subject

Endocrinology,Molecular Biology,Biochemistry

Reference42 articles.

1. Metabolic stress-induced activation of FoxO1 triggers diabetic cardiomyopathy in mice;Battiprolu;J. Clin. Invest.,2012

2. Diabetic cardiomyopathy and subclinical cardiovascular disease: the Multi-Ethnic Study of Atherosclerosis (MESA);Bertoni;Diabetes Care,2006

3. Reference gene alternatives to Gapdh in rodent and human heart failure gene expression studies;Brattelid;BMC Mol. Biol.,2010

4. Protein kinase SGK mediates survival signals by phosphorylating the forkhead transcription factor FKHRL1 (FOXO3a);Brunet;Mol. Cell Biol.,2001

5. The FoxO code;Calnan;Oncogene,2008

Cited by 12 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Maternal diabetes increases FOXO1 activation during embryonic cardiac development;Molecular and Cellular Endocrinology;2023-09

2. Weighted gene co-expression network analysis reveals similarities and differences of molecular features between dilated and ischemic cardiomyopathies;Journal of Electronic Science and Technology;2023-06

3. Intrauterine Programming of Cardiovascular Diseases in Maternal Diabetes;Frontiers in Physiology;2021-11-03

4. High-Mobility Group A1 Promotes Cardiac Fibrosis by Upregulating FOXO1 in Fibroblasts;Frontiers in Cell and Developmental Biology;2021-08-26

5. FoxO1 is required for high glucose-dependent cardiac fibroblasts into myofibroblast phenoconversion;Cellular Signalling;2021-07

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