Overexpression of Lin28a induces a primary ovarian insufficiency phenotype via facilitation of primordial follicle activation in mice
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Molecular Biology,Biochemistry
Reference49 articles.
1. WNT5a is required for normal ovarian follicle development and antagonizes gonadotropin responsiveness in granulosa cells by suppressing canonical WNT signaling;Abedini;Faseb. J.,2016
2. Disruption of Tsc2 in oocytes leads to overactivation of the entire pool of primordial follicles;Adhikari;Mol. Hum. Reprod.,2009
3. Tsc/mTORC1 signaling in oocytes governs the quiescence and activation of primordial follicles;Adhikari;Hum. Mol. Genet.,2010
4. Mutations and mechanisms of WNT pathway tumour suppressors in cancer;Bugter;Nat. Rev. Canc.,2021
5. The Wnt–β-catenin pathway represses let-7 microRNA expression through transactivation of Lin28 to augment breast cancer stem cell expansion;Cai;J. Cell Sci.,2013
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