Branched-chain amino acid catabolic defect promotes α-cell proliferation via activating mTOR signaling
Author:
Funder
National Natural Science Foundation of China
Science and Technology Commission of Shanghai Municipality
Publisher
Elsevier BV
Subject
Endocrinology,Molecular Biology,Biochemistry
Reference50 articles.
1. Metabolic effects of glucagon in humans;Adeva-Andany;J. Clin. Transl. Endocrinol.,2019
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3. Branched-chain amino acid catabolism in muscle affects systemic BCAA levels but not insulin resistance;Blair;Nat. Metab.,2023
4. Loss of mTORC1 signaling alters pancreatic α-cell mass and impairs glucagon secretion;Bozadjieva;J. Clin. Invest.,2017
5. SAR1B senses leucine levels to regulate mTORC1 signalling;Chen;Nature,2021
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