CaMKII(δ) regulates osteoclastogenesis through ERK, JNK, and p38 MAPKs and CREB signalling pathway-Reference-Cited by-同舟云学术

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CaMKII(δ) regulates osteoclastogenesis through ERK, JNK, and p38 MAPKs and CREB signalling pathway

Published:2020-05 Issue: Volume:508 Page:110791
ISSN:0303-7207
Container-title:Molecular and Cellular Endocrinology
language:en
Short-container-title:Molecular and Cellular Endocrinology

Author:

Lu Da-Zhuang,Dong Wei,Feng Xiao-Jie,Chen Hui,Liu Juan-Juan,Wang Hui,Zang Lu-Yang,Qi Meng-Chun

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Endocrinology,Molecular Biology,Biochemistry

Reference36 articles.

1. Calcium/calmodulin-dependent kinase activity is required for efficient induction of osteoclast differentiation and bone resorption by receptor activator of nuclear factor kappa B ligand (RANKL);Ang;J. Cell. Physiol.,2007

2. A RANK/TRAF6-dependent signal transduction pathway is essential for osteoclast cytoskeletal organization and resorptive function;Armstrong;J. Biol. Chem.,2002

3. Auto-amplification of NFATc1 expression determines its essential role in bone homeostasis;Asagiri;J. Exp. Med.,2005

4. The JNK-dependent CaMK pathway restrains the reversion of committed cells during osteoclast differentiation;Chang;J. Cell Sci.,2008

5. Ameloblastin attenuates RANKL-mediated osteoclastogenesis by suppressing activation of nuclear factor of activated T-cell cytoplasmic 1 (NFATc1);Chaweewannakorn;J. Cell. Physiol.,2019

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