High glucose up-regulates Semaphorin 3A expression via the mTOR signaling pathway in keratinocytes: A potential mechanism and therapeutic target for diabetic small fiber neuropathy

Author:

Wu Liang-yanORCID,Li MeiORCID,Qu Min-LiORCID,Li XinORCID,Pi Lin-HuaORCID,Chen ZiORCID,Zhou Shan-LeiORCID,Yi Xiao-QingORCID,Shi Xia-Jie,Wu Jing,Wang Shan

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Endocrinology,Molecular Biology,Biochemistry

Reference59 articles.

1. Semaphorin3a promotes advanced diabetic nephropathy;Aggarwal;Diabetes,2015

2. Mammalian target of rapamycin signaling in the spinal cord is required for neuronal plasticity and behavioral hypersensitivity associated with neuropathy in the rat;Asante;J. Pain,2010

3. mTORC1 promotes proliferation of immature Schwann cells and myelin growth of differentiated Schwann cells;Beirowski;Proc. Natl. Acad. Sci. U. S. A.,2017

4. Semaphorin3A regulates axon growth independently of growth cone repulsion via modulation of TrkA signaling;Ben-Zvi;Cell SIGNAL,2008

5. Neuron-derived semaphorin 3A is an early inducer of vascular permeability in diabetic retinopathy via neuropilin-1;Cerani;Cell Metab.,2013

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