SIRT1 suppresses high glucose and palmitate-induced osteoclast differentiation via deacetylating p66Shc-Reference-Cited by-同舟云学术

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SIRT1 suppresses high glucose and palmitate-induced osteoclast differentiation via deacetylating p66Shc

Published:2018-10 Issue: Volume:474 Page:97-104
ISSN:0303-7207
Container-title:Molecular and Cellular Endocrinology
language:en
Short-container-title:Molecular and Cellular Endocrinology

Author:

Qu Bo,Gong Kai,Yang Hongsheng,Li Yugang,Jiang Tao,Zeng Zhimou,Cao Zongrui,Pan Xianming

Funder

China Postdoctoral Science Foundation

Sichuan Provincial Science and Technology Department

Sichuan Medical Association

Publisher

Elsevier BV

Subject

Endocrinology,Molecular Biology,Biochemistry

Reference35 articles.

1. Oxidative stress stimulates apoptosis and activates NF-kappaB in osteoblastic cells via a PKCbeta/p66shc signaling cascade: counter regulation by estrogens or androgens;Almeida;Mol. Endocrinol.,2010

2. Free fatty acids, insulin resistance, and type 2 diabetes mellitus;Boden;Proc Assoc Am Physicians,1999

3. Hyperglycemia induced and intrinsic alterations in type 2 diabetes-derived osteoclast function;Catalfamo;Oral Dis.,2013

4. Palmitic acid and DGAT1 deficiency enhance osteoclastogenesis, while oleic acid-induced triglyceride formation prevents it;Drosatos-Tampakaki;J. Bone Miner. Res.,2014

5. Palmitic acid increases pro-oxidant adaptor protein p66Shc expression and affects vascularization factors in angiogenic mononuclear cells: action of resveratrol;Favre;Vascul Pharmacol,2015

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