Reciprocal regulation between GCN2 (eIF2AK4) and PERK (eIF2AK3) through the JNK-FOXO3 axis to modulate cancer drug resistance and clonal survival-Reference-Cited by-同舟云学术

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Reciprocal regulation between GCN2 (eIF2AK4) and PERK (eIF2AK3) through the JNK-FOXO3 axis to modulate cancer drug resistance and clonal survival

Published:2020-09 Issue: Volume:515 Page:110932
ISSN:0303-7207
Container-title:Molecular and Cellular Endocrinology
language:en
Short-container-title:Molecular and Cellular Endocrinology

Author:

Alasiri Glowi,Jiramongkol Yannasittha,Trakansuebkul Sasanan,Ke Hui-Ling,Mahmud Zimam,Intuyod Kitti,Lam Eric W.-F.

Funder

Commonwealth Scholarship Commission

Breast Cancer Now

Saudi Arabian Cultural Mission

Cancer Research UK

Medical Research Council

Thailand Research Fund

Publisher

Elsevier BV

Subject

Endocrinology,Molecular Biology,Biochemistry

Reference44 articles.

1. ER stress and cancer: the FOXO forkhead transcription factor link;Alasiri;Mol. Cell. Endocrinol.,2018

2. Regulation of PERK expression by FOXO3: a vulnerability of drug-resistant cancer cells;Alasiri;Oncogene,2019

3. Characterization of a novel PERK kinase inhibitor with antitumor and antiangiogenic activity;Atkins;Canc. Res.,2013

4. Discovery of 7-methyl-5-(1-{[3-(trifluoromethyl)phenyl]acetyl}-2,3-dihydro-1H-indol-5-yl)-7H-p yrrolo[2,3-d]pyrimidin-4-amine (GSK2606414), a potent and selective first-in-class inhibitor of protein kinase R (PKR)-like endoplasmic reticulum kinase (PERK);Axten;J. Med. Chem.,2012

5. Discovery of GSK2656157: an optimized PERK inhibitor selected for preclinical development;Axten;ACS Med. Chem. Lett.,2013

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