The mechanism of 14-3-3η in thyroxine induced mitophagy in cardiomyocytes-Reference-Cited by-同舟云学术

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The mechanism of 14-3-3η in thyroxine induced mitophagy in cardiomyocytes

Published:2024-09 Issue: Volume:590 Page:112271
ISSN:0303-7207
Container-title:Molecular and Cellular Endocrinology
language:en
Short-container-title:Molecular and Cellular Endocrinology

Author:

Cui Yalan,Zhang Yan,Dai Songsong,Wan Sha,Guan Heng,Wang Decai,Jin Beifang,Xiao Wenping,Liu Fang

Funder

National Natural Science Foundation of China

Guilin Medical University

Publisher

Elsevier BV

Reference56 articles.

1. Mitochondrial fusion and fission: the fine‐tune balance for cellular homeostasis;Adebayo;Faseb. J.,2021

2. Hypothyroidism and spine surgery: a review of current findings;Bajaj;Curr Rev Musculoskelet Med,2023

3. N-acetylcysteine, an ROS inhibitor, alleviates the pathophysiology of hyperthyroidism-induced cardiomyopathy via the ROS/Ca(2+) pathway;Bao;Biomolecules,2022

4. Impact of hyperthyroidism on cardiac hypertrophy;Barreto-Chaves;Endocr Connect,2020

5. 14-3-3 proteins in the nervous system;Berg;Nat. Rev. Neurosci.,2003

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Regulation of H9C2 cell hypertrophy by 14-3-3η via inhibiting glycolysis;PLOS ONE;2024-07-22

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