Regulation of endocannabinoid release by G proteins: A paracrine mechanism of G protein-coupled receptor action-Reference-Cited by-同舟云学术

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Regulation of endocannabinoid release by G proteins: A paracrine mechanism of G protein-coupled receptor action

Published:2012-04 Issue:1-2 Volume:353 Page:29-36
ISSN:0303-7207
Container-title:Molecular and Cellular Endocrinology
language:en
Short-container-title:Molecular and Cellular Endocrinology

Author:

Gyombolai Pál,Pap Dorottya,Turu Gábor,Catt Kevin J.,Bagdy György,Hunyady László

Funder

Hungarian Science Foundation

the Hungarian Ministry of Public Health

ETT 318/2009

Publisher

Elsevier BV

Subject

Endocrinology,Molecular Biology,Biochemistry

Reference89 articles.

1. Supply and demand for endocannabinoids;Alger;Trends Neurosci.,2011

2. Lack of CB1 receptor activity impairs serotonergic negative feedback;Aso;J. Neurochem.,2009

3. Long-lasting self-inhibition of neocortical interneurons mediated by endocannabinoids;Bacci;Nature,2004

4. Role of the hypothalamic paraventricular nucleus in 5-HT1A, 5-HT2A and 5-HT2C receptor-mediated oxytocin, prolactin and ACTH/corticosterone responses;Bagdy;Behav. Brain Res.,1996

5. Hypothalamic paraventricular nucleus lesions differentially affect serotonin-1A (5-HT1A) and 5-HT2 receptor agonist-induced oxytocin, prolactin, and corticosterone responses;Bagdy;Endocrinology,1994

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