Differential control of uterine artery endothelial monolayer integrity by TNF and VEGF is achieved through multiple mechanisms operating inside and outside the cell – Relevance to preeclampsia
Author:
Funder
NIH
Eunice Kennedy Shriver National Institute of Child Health and Human Development
Publisher
Elsevier BV
Subject
Endocrinology,Molecular Biology,Biochemistry
Reference15 articles.
1. TNF-alpha inhibits pregnancy-adapted Ca2+ signaling in uterine artery endothelial cells;Ampey;Mol. Cell. Endocrinol.,2019
2. Membrane-anchored proteases in endothelial cell biology;Antalis;Curr. Opin. Hematol.,2016
3. Pregnancy, programming and preeclampsia: gap junctions at the nexus of pregnancy-induced adaptation of endothelial function and endothelial adaptive failure in PE;Bird;Curr. Vasc. Pharmacol.,2013
4. Phosphorylation of Ser-279/282 and Tyr-265 positions on Cx43 as possible mediators of VEGF-165 inhibition of pregnancy-adapted Ca2+ burst function in ovine uterine artery endothelial cells;Boeldt;Mol. Cell. Endocrinol.,2015
5. Positive versus negative effects of VEGF165 on Ca2+ signaling and NO production in human endothelial cells;Boeldt;Am. J. Physiol. Heart Circ. Physiol.,2017
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