Mof acetyltransferase inhibition ameliorates glucose intolerance and islet dysfunction of type 2 diabetes via targeting pancreatic α-cells
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Molecular Biology,Biochemistry
Reference48 articles.
1. Glucagon antagonism as a potential therapeutic target in type 2 diabetes;Bagger;Diabetes Obes. Metabol.,2011
2. Loss of mTORC1 signaling alters pancreatic α cell mass and impairs glucagon secretion;Bozadjieva;J. Clin. Invest.,2017
3. MOF acetyl transferase regulates transcription and respiration in mitochondria;Chatterjee;Cell,2016
4. Histone deacetylase (HDAC) inhibition as a novel treatment for diabetes mellitus;Christensen;Mol. Med.,2011
5. Glucagon receptor knockout mice are resistant to diet-induced obesity and streptozotocin-mediated beta cell loss and hyperglycaemia;Conarello;Diabetologia,2007
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1. Personalized epigenetic management of diabetes;Personalized Epigenetics;2024
2. Mof plays distinct roles in hepatic lipid metabolism under healthy or non-alcoholic fatty liver conditions;iScience;2023-12
3. cGAS‐STING mediates cytoplasmic mitochondrial‐DNA‐induced inflammatory signal transduction during accelerated senescence of pancreatic β‐cells induced by metabolic stress;The FASEB Journal;2022-03-31
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