APPL1-mediated activation of STAT3 contributes to inhibitory effect of adiponectin on hepatic gluconeogenesis-Reference-Cited by-同舟云学术

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APPL1-mediated activation of STAT3 contributes to inhibitory effect of adiponectin on hepatic gluconeogenesis

Published:2016-09 Issue: Volume:433 Page:12-19
ISSN:0303-7207
Container-title:Molecular and Cellular Endocrinology
language:en
Short-container-title:Molecular and Cellular Endocrinology

Author:

Ding Youming,Zhang Deling,Wang Bin,Zhang Yemin,Wang Lei,Chen Xiaoyan,Li Mingxin,Tang Zhao,Wang Changhua

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Endocrinology,Molecular Biology,Biochemistry

Reference41 articles.

1. Negative regulation of STAT3 protein-mediated cellular respiration by SIRT1 protein;Bernier;J. Biol. Chem.,2011

2. Adiponectin blocks interleukin-18-mediated endothelial cell death via APPL1-dependent AMP-activated protein kinase (AMPK) activation and IKK/NF-kappaB/PTEN suppression;Chandrasekar;J. Biol. Chem.,2008

3. Adiponectin-induced endothelial nitric oxide synthase activation and nitric oxide production are mediated by APPL1 in endothelial cells;Cheng;Diabetes,2007

4. APPL1 potentiates insulin-mediated inhibition of hepatic glucose production and alleviates diabetes via Akt activation in mice;Cheng;Cell Metab.,2009

5. APPL1 potentiates insulin secretion in pancreatic β cells by enhancing protein kinase Akt-dependent expression of SNARE proteins in mice;Cheng;Proc. Natl. Acad. Sci. U. S. A.,2012

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