Diesel Exhaust Particles Increase NF-κB DNA Binding Activity and c-FOS Proto-oncogene Expression in Human Bronchial Epithelial Cells

Author:

Baeza-Squiban A.,Bonvallot V.,Boland S.,Marano F.

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference19 articles.

1. Cyclosporin A-sensitive calcium signalling represses NFκB activation in human bronchial epithelial cells and enhances NFκB activation in Jurkat T-cells;Aoki;Biochemical and Biophysical Research Communications,1997

2. The effects of diesel exhaust particles on cell function and release of inflammatory mediators from human bronchial epithelial cells in vitro;Bayram;American Journal of Respiratory Cell and Molecular Biology,1998

3. Primary cultures of tracheal epithelial cells for the evaluation of respiratory toxicity;Blanquart;Toxicology in Vitro,1991

4. Diesel exhaust particles are phagocyted by human airway epithelial cells in vitro and alter cytokine production;Boland;American Journal of Physiology,1999

5. Boland S., Baeza-Squiban A., Houcine O., Guennou C. and Marano F. (1998) Effects of diesel exhaust particles on the viability and morphology of airway epithelial cells in primary culture. In Relationships Between Respiratory Disease and Exposure to Air Pollution. Edited by D. L. Dungworth, J. D. Brain, K. E. Driscoll, R. C. Grafström and C. C. Harris, pp. 273–277. ILSI Press, Washington

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