Human cardiac microvascular endothelial cells/α1-agonists/endothelial dysfunction: pathophysiologic connotations for takotsubo syndrome-Reference-Cited by-同舟云学术

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Human cardiac microvascular endothelial cells/α1-agonists/endothelial dysfunction: pathophysiologic connotations for takotsubo syndrome

Published:2024-09 Issue: Volume:155 Page:104717
ISSN:0026-2862
Container-title:Microvascular Research
language:en
Short-container-title:Microvascular Research

Author:

Madias John E.

Publisher

Elsevier BV

Reference7 articles.

1. Abnormal thyroid function is common in takotsubo syndrome and depends on two distinct mechanisms: results of a multicentre observational study;Aweimer;J. Intern. Med.,2021

2. Takotsubo syndrome outcomes predicted by thyroid hormone signature: insights from cluster analysis of a multicentre registry;Aweimer;EBioMedicine,2024

3. Catecholamine-dependent beta-adrenergic signaling in a pluripotent stem cell model of takotsubo cardiomyopathy;Borchert;J. Am. Coll. Cardiol.,2017

4. An update on the mechanisms of Takotsubo syndrome: “at the end an acute coronary syndrome”;Crea;J. Mol. Cell. Cardiol.,2024

5. Alpha 1 adrenoceptor signaling contributes to toxic effects of catecholamine on electrical properties in human-induced pluripotent stem cell derived cardiomyocytes;Huang;Europace,2021

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