Vascular leakage induced by exposure to arsenic via increased production of NO, hydroxyl radical and peroxynitrite

Author:

Chen Shih-Chieh,Chen Wei-Chi

Publisher

Elsevier BV

Subject

Cell Biology,Cardiology and Cardiovascular Medicine,Biochemistry

Reference38 articles.

1. Inhibition of nitric oxide synthesis increases venular permeability and alters endothelial actin cytoskeleton;Baldwin;Am. J. Physiol.,1998

2. Stimulation of reactive oxygen, but not reactive nitrogen species, in vascular endothelial cells exposed to low levels of arsenite;Barchowsky;Free Radic. Biol. Med.,1999

3. Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide;Beckman;Proc. Natl. Acad. Sci. U. S. A.,1990

4. Evidence for participation of hydroxyl radical in increased microvascular permeability;Bjork;Agents Actions Suppl.,1980

5. Cancer potential in liver, lung, bladder and kidney due to ingested inorganic arsenic in drinking water;Chen;Br. J. Cancer.,1992

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