p27 Kip1 and p21 Cip1 -independent proliferative inhibition of vascular smooth muscle cells cultured in type-I collagen matrix honeycombs

Author:

Uchida Masashi,Suzuki Saki,Suzuki Takaaki,Ishii Itsuko

Funder

The Research Foundation for Pharmaceutical Sciences

Publisher

Elsevier BV

Subject

Cell Biology,Cardiology and Cardiovascular Medicine,Biochemistry

Reference25 articles.

1. Adenovirus-mediated over-expression of the cyclin/cyclin-dependent kinase inhibitor, p21 inhibits vascular smooth muscle cell proliferation and neointima formation in the rat carotid artery model of balloon angioplasty;Chang;J. Clin. Invest.,1995

2. Magae, mdm2 and bax, downstream mediators of the p53 response, are degraded by the ubiquitin–proteasome pathway;Chang;Cell Growth Differ.,1998

3. Downregulation of cyclin-dependent kinase 2 activity and cyclin A promoter activity in vascular smooth muscle cells by p27(KIP1), an inhibitor of neointima formation in the rat carotid artery;Chen;J. Clin. Invest.,1997

4. Kinetics of cellular proliferation after arterial injury. I. Smooth muscle cell growth in the absence of endothelium;Clowes;Lab. Investig.,1983

5. Time course of cellular proliferation, intimal hyperplasia, and remodeling following angioplasty in monkeys with established atherosclerosis. A nonhuman primate model of restenosis;Geary;Arterioscler. Thromb. Vasc. Biol.,1996

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