GPX4 allosteric activators inhibit ferroptosis and exert myocardial protection in doxorubicin-induced myocardial injury mouse model-Reference-Cited by-同舟云学术

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GPX4 allosteric activators inhibit ferroptosis and exert myocardial protection in doxorubicin-induced myocardial injury mouse model

Published:2024-11 Issue: Volume:277 Page:116721
ISSN:0223-5234
Container-title:European Journal of Medicinal Chemistry
language:en
Short-container-title:European Journal of Medicinal Chemistry

Author:

Liu Xiaoang^ORCID,Guo Yi,Huang Yusong,Wang Qian,Huang Yanwen,Lei Yizhi,Liu Zhenming,Zhang Liangren

Funder

National Natural Science Foundation of China

National Key Research and Development Program of China

Publisher

Elsevier BV

Reference41 articles.

1. Ferroptosis: an iron-dependent form of nonapoptotic cell death;Dixon;Cell,2012

2. Targeting a novel inducible GPX4 alternative isoform to alleviate ferroptosis and treat metabolic-associated fatty liver disease;Tong;Acta Pharm. Sin. B,2022

3. Boosting synergism of chemo- and immuno-therapies via switching paclitaxel-induced apoptosis to mevalonate metabolism-triggered ferroptosis by bisphosphonate coordination lipid nanogranules;Song;Acta Pharm. Sin. B,2024

4. Sorting nexin 3 exacerbates doxorubicin-induced cardiomyopathy via regulation of TFRC-dependent ferroptosis;Yu;Acta Pharm. Sin. B,2023

5. Ferroptosis is involved in acetaminophen induced cell death;Lőrincz;Pathol. Oncol. Res.,2015

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