Triptolide induced spermatogenesis dysfunction via ferroptosis activation by promoting K63-linked GPX4 polyubiquitination in spermatocytes-Reference-Cited by-同舟云学术

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Triptolide induced spermatogenesis dysfunction via ferroptosis activation by promoting K63-linked GPX4 polyubiquitination in spermatocytes

Published:2024-08 Issue: Volume:399 Page:111130
ISSN:0009-2797
Container-title:Chemico-Biological Interactions
language:en
Short-container-title:Chemico-Biological Interactions

Author:

Li Jiaqi,Chen Dezhi,Suo Jialiang,Li Jiaqi,Zhang Yimu,Wang Yu,Deng Zhewen,Zhang Qi,Ma Bo^ORCID

Publisher

Elsevier BV

Reference49 articles.

1. A mechanistic overview of triptolide and celastrol, natural products from tripterygium wilfordii Hook F;Chen;Front. Pharmacol.,2018

2. Immunoregulatory effects of Tripterygium wilfordii Hook F and its extracts in clinical practice;Luo;Front. Med.,2019

3. Toxicity of triptolide and the molecular mechanisms involved;Xi;Biomed. Pharmacother.,2017

4. MitoQ ameliorates testis injury from oxidative attack by repairing mitochondria and promoting the Keap1-Nrf2 pathway;Zhang;Toxicol. Appl. Pharmacol.,2019

5. A natural iridoid glucoside, attenuates oxidative stress-induced testis injury by inhibiting JNK and CHOP activation via Nrf2 up-regulation;Ma;Phytomedicine,2019

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