Exogenous iron caused osteocyte apoptosis, increased RANKL production, and stimulated bone resorption through oxidative stress in a murine model-Reference-Cited by-同舟云学术

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Exogenous iron caused osteocyte apoptosis, increased RANKL production, and stimulated bone resorption through oxidative stress in a murine model

Published:2024-08 Issue: Volume:399 Page:111135
ISSN:0009-2797
Container-title:Chemico-Biological Interactions
language:en
Short-container-title:Chemico-Biological Interactions

Author:

Guo Zengfeng,Wu Jiawen,Hu Yawei,Zhou Jianhua,Li Qingmei,Zhang Yandong,Zhang Junde,Yang Linbo,Wang Shenghang,Zhang Hao,Yang Jiancheng^ORCID

Publisher

Elsevier BV

Reference45 articles.

1. Iron metabolism: pathophysiology and pharmacology;Roemhild;Trends Pharmacol. Sci.,2021

2. The effect of abnormal iron metabolism on osteoporosis;Che;Biol. Trace Elem. Res.,2020

3. Clinical impact and cellular mechanisms of iron overload-associated bone loss;Jeney;Front. Pharmacol.,2017

4. Bone loss caused by iron overload in a murine model: importance of oxidative stress;Tsay;Blood,2010

5. Novel and rapid osteoporosis model established in zebrafish using high iron stress;Zhang;Biochem. Biophys. Res. Commun.,2018

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