Effects of dietary butylated hydroxytoluene and phenobarbital on the activities of ornithine decarboxylase and thymidine kinase in rat liver and lung

Author:

Saccone G.T.P.,Pariza M.W.

Publisher

Elsevier BV

Subject

Cancer Research,Oncology

Reference15 articles.

1. The function and mechanism of promoters of carcinogenesis;Boutwell;Crit. Rev. Toxicol.,1974

2. Activation of 3′:5′-cyclic AMP-dependent protein kinase and induction of ornithine decarboxylase as early events in induction of mixed-function oxygenases;Byus,1976

3. Food and light as separate entrainment signals for rat liver enzymes;Hopkins;Adv. Enzyme Regul.,1973

4. Thymidine kinase and deoxyribonucleic acid metabolism in growing and regenerating livers from rats on controlled feeding schedules;Hopkins;Biochem. J.,1973

5. Induction of the polyamine biosynthetic enzymes in mouse epidermis by tumor-promoting agents;O'Brien;Cancer Res.,1975

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1. Final Report on the Safety Assessment of BHT;International Journal of Toxicology;2002-10

2. Enhancement of biliary excretion of aflatoxin B1 and suppression of hepatic ornithine decarboxylase activity by 2-(allylthio)pyrazine in rats;Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis;1999-07

3. Liver pyruvate kinase assay as a predicting screening test for promoters of hepatocarcinogenesis in the rat;Journal of Environmental Science and Health, Part C;1994-05

4. METABOLISM AND PULMONARY TOXICITY OF BUTYLATED HYDROXYTOLUENE;Metabolic Activation and Toxicity of Chemical Agents to Lung Tissue and Cells;1993

5. Toxicological Aspects of Antioxidants Used as Food Additives;Food Antioxidants;1990

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