iNOS contributes to heart failure with preserved ejection fraction through mitochondrial dysfunction and Akt S-nitrosylation-Reference-Cited by-同舟云学术

iNOS contributes to heart failure with preserved ejection fraction through mitochondrial dysfunction and Akt S-nitrosylation

Published:2023-01 Issue: Volume:43 Page:175-186
ISSN:2090-1232
Container-title:Journal of Advanced Research
language:en
Short-container-title:Journal of Advanced Research

Author:

Guo Yongzheng,Wen Junjie,He An,Qu Can,Peng Yuce,Luo Suxin,Wang Xiaowen

Publisher

Elsevier BV

Subject

Multidisciplinary

Reference34 articles.

1. Heart disease and Stroke Statistics-2019 Update: A Report From the American Heart Association;Benjamin;Circulation,2019

2. Chronic low-grade inflammation in heart failure with preserved ejection fraction;Mesquita;Aging Cell,2021

3. Association between low-grade chronic inflammation and depressed left atrial compliance in heart failure with preserved ejection fraction: A retrospective analysis;Sani;Folia Med Cracov,2018

4. Heart Failure with Preserved Ejection Fraction: Current Opinion and Future Perspectives;Hong;Heart Fail Clin,2021

5. Mitochondrial Dysfunction in Heart Failure With Preserved Ejection Fraction;Kumar;Circulation,2019

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3. Valosin-containing protein acts as a target and mediator of S-nitrosylation in the heart through distinct mechanisms;Redox Biology;2024-06

4. INOS ablation promotes corneal wound healing via activation of Akt signaling;Experimental Eye Research;2024-06

5. The total xanthones extracted from Gentianella acuta alleviates HFpEF by activating the IRE1α/Xbp1s pathway;Journal of Cellular and Molecular Medicine;2024-06