Slit2/Robo4 signaling pathway modulates endothelial hyper-permeability in a two-event in vitro model of transfusion-related acute lung injury-Reference-Cited by-同舟云学术

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Slit2/Robo4 signaling pathway modulates endothelial hyper-permeability in a two-event in vitro model of transfusion-related acute lung injury

Published:2019-05 Issue: Volume:76 Page:7-12
ISSN:1079-9796
Container-title:Blood Cells, Molecules, and Diseases
language:en
Short-container-title:Blood Cells, Molecules, and Diseases

Author:

Weng Jie,Zhou Xiaoming,Xie Hui,Gao Ye,Wang Zhiyi,Gong Yuqiang

Funder

Zhejiang Provincial Natural Science Foundation

Publisher

Elsevier BV

Subject

Cell Biology,Hematology,Molecular Biology,Molecular Medicine

Reference34 articles.

1. Transfusion-related acute lung injury: a dangerous and underdiagnosed noncardiogenic pulmonary edema;Jaworski;Cardiol. J.,2013

2. Transfusion-related acute lung injury;El Kenz;Eur. J. Anaesthesiol.,2014

3. Transfusion-related acute lung injury: transfusion, platelets and biological response modifiers;Tariket;Expert. Rev. Hematol.,2016

4. Transfusion-related acute lung injury not a two-hit, but a multicausal model;Middelburg;Transfusion,2015

5. Transfusion-related acute lung injury: from bedside to bench and back;Shaz;Blood,2011

Cited by 13 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Slit2–Robo4 signal pathway and tight junction in intestine mediate LPS-induced inflammation in mice;European Journal of Medical Research;2024-06-27

2. Attenuation of Transfusion-Related Acute Lung Injury by FAK Inhibitor PF-562271;J BIOL REG HOMEOS AG;2023

3. Recombinant Slit2 suppresses neuroinflammation and Cdc42-mediated brain infiltration of peripheral immune cells via Robo1–srGAP1 pathway in a rat model of germinal matrix hemorrhage;Journal of Neuroinflammation;2023-10-29

4. Attenuation of transfusion-related acute lung injury by FAK inhibitor PF-562271;2023-06-08

5. Slit2-Robo4 Signal Pathway and Tight Junction in Intestine Mediate LPS-Induced Inflammation in Mice;2023-04-06

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