Genetic modifiers of secondary iron overload in beta thalassemia major-Reference-Cited by-同舟云学术

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Genetic modifiers of secondary iron overload in beta thalassemia major

Published:2015-03 Issue:3 Volume:54 Page:242-243
ISSN:1079-9796
Container-title:Blood Cells, Molecules, and Diseases
language:en
Short-container-title:Blood Cells, Molecules, and Diseases

Author:

Athiyarath Rekha,George Biju,Abraham Aby,Viswabandya Auro,Srivastava Alok,Edison Eunice Sindhuvi

Funder

Department of Biotechnology, Government of India

Publisher

Elsevier BV

Subject

Cell Biology,Hematology,Molecular Biology,Molecular Medicine

Reference8 articles.

1. β-Thalassemia: hiJAKing ineffective erythropoiesis and iron overload;Melchiori;Adv. Hematol.,2010

2. A novel missense mutation in SLC40A1 results in resistance to hepcidin and confirms the existence of two ferroportin-associated iron overload diseases;Létocart;Br. J. Haematol.,2009

3. Association of hepcidin promoter c.-582 A>G variant and iron overload in thalassemia major;Andreani;Haematologica,2009

4. Hepcidin mutation in a beta-thalassemia major patient with persistent severe iron overload despite chelation therapy;Duca;Intern. Emerg. Med.,2010

5. HFE mutation H63D predicts risk of iron over load in thalassemia intermedia irrespective of blood transfusions;Sharma;Indian J. Pathol. Microbiol.,2007

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Calprotectin;Medical Research Journal;2015-12

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