Hexose metabolism in pancreatic islets. Ca(2+)-dependent activation of the glycerol phosphate shuttle by nutrient secretagogues.
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference17 articles.
1. Hexose metabolism in pancreatic islets. Regulation of aerobic glycolysis and pyruvate decarboxylation
2. High content of mitochondrial glycerol-3-phosphate dehydrogenase in pancreatic islets and its inhibition by diazoxide.
3. Calcium Activation of Pancreatic Islet Mitochondrial Glycerol Phosphate Dehydrogenase
4. Hexose metabolism in pancreatic islets. Glucose-induced and Ca2+-dependent activation of FAD-glycerophosphate dehydrogenase
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1. Effects of plasma membrane Ca2+-ATPase overexpression upon D-glucose metabolism in insulin-producing BRIN-BD11 cells;Bioscience Reports;2008-09-11
2. Glucose-sensing mechanisms in pancreatic β-cells;Philosophical Transactions of the Royal Society B: Biological Sciences;2005-11-04
3. Mitochondrial Metabolism Sets the Maximal Limit of Fuel-stimulated Insulin Secretion in a Model Pancreatic Beta Cell;Journal of Biological Chemistry;2002-04
4. Site-Directed Mutations in the FAD-Binding Domain of Glycerophosphate Dehydrogenase: Catalytic Defects with Preserved Mitochondrial Anchoring of the Enzyme in Transfected COS-7 Cells;Molecular Genetics and Metabolism;2002-02
5. Redox regulation of cytosolic glycerol-3-phosphate dehydrogenase: Cys102 is the target of the redox control and essential for the catalytic activity;Biochimica et Biophysica Acta (BBA) - General Subjects;2002-01
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