Raf-1 kinase activity is necessary and sufficient for gene expression changes but not sufficient for cellular morphology changes associated with cardiac myocyte hypertrophy.-Reference-Cited by-同舟云学术

Raf-1 kinase activity is necessary and sufficient for gene expression changes but not sufficient for cellular morphology changes associated with cardiac myocyte hypertrophy.

Published:1994-12 Issue:48 Volume:269 Page:30580-30586
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Thorburn J,McMahon M,Thorburn A

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference43 articles.

1. Regulation of cardiac gene expression during myocardial growth and hypertrophy: molecular studies of an adaptive physiologic response

2. Norepinephrine-stimulated hypertrophy of cultured rat myocardial cells is an alpha 1 adrenergic response.

3. Protooncogene induction and reprogramming of cardiac gene expression produced by pressure overload.

4. Co-regulation of the atrial natriuretic factor and cardiac myosin light chain-2 genes during alpha-adrenergic stimulation of neonatal rat ventricular cells. Identification of cis sequences within an embryonic and a constitutive contractile protein gene which mediate inducible expression.

5. Alpha 1-adrenergic stimulation of cardiac gene transcription in neonatal rat myocardial cells. Effects on myosin light chain-2 gene expression.

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1. Differential effects of Pyk2 and FAK on the hypertrophic response of cardiac myocytes;Cell and Tissue Research;2009-05-12

2. With great power comes great responsibility: Using mouse genetics to study cardiac hypertrophy and failure;Journal of Molecular and Cellular Cardiology;2009-02

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4. Genetic inhibition of cardiac ERK1/2 promotes stress-induced apoptosis and heart failure but has no effect on hypertrophy in vivo;Proceedings of the National Academy of Sciences;2007-08-20

5. Ras triggers acidosis-induced activation of the extracellular-signal-regulated kinase pathway in cardiac myocytes;Biochemical Journal;2006-10-13