Inhibition of atrial natriuretic peptide-induced cGMP accumulation by purinergic agonists in FRTL-5 thyroid cells. Involvement of both pertussis toxin-sensitive and insensitive mechanisms.
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference38 articles.
1. Is there a basis for distinguishing two types of P2-purinoceptor?
2. Extracellular ATP: effects, sources and fate
3. Inhibition by Islet-Activating Protein, Pertussis Toxin, of P2-Purinergic Receptor-Mediated Iodide Efflux and Phosphoinositide Turnover in FRTL-5 Cells*
4. A Permissive Role of Pertussis Toxin Substrate G-protein in P2-purinergic Stimulation of Phosphoinositide Turnover and Arachidonate Release in FRTL-5 Thyroid Cells
5. Islet-activating protein, pertussis toxin: a probe for functions of the inhibitory guanine nucleotide regulatory component of adenylate cyclase
Cited by 15 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. An adenosine receptor agonist-induced modulation of TSH-dependent cell growth in FRTL-5 thyroid cells mediated by inhibitory G protein, Gi;Biochimie;1999-04
2. Extracellular adenosine 5′-triphosphate induces Ca2+ efflux from freshly isolated adult rat cardiomyocytes: Possible involvement of exchange mechanism;Life Sciences;1997-09
3. βγ Subunits of Pertussis Toxin-sensitive G Proteins Mediate A1 Adenosine Receptor Agonist-induced Activation of Phospholipase C in Collaboration with Thyrotropin;Journal of Biological Chemistry;1997-09
4. Opposing adenine nucleotide-dependent pathways regulate guanylyl cyclase C in rat intestine.;Journal of Biological Chemistry;1994-09
5. Effect of PT-treatment on ANP-mediated inhibition of adenylate cyclase and amylase release in rat parotid gland;Molecular and Cellular Biochemistry;1994
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