Chemotactic Peptide Induces cAMP Elevation in Human Neutrophils by Amplification of the Adenylate Cyclase Response to Endogenously Produced Adenosine
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference23 articles.
1. Increased levels of cyclic adenosine-3',5'-monophosphate in human polymorphonuclear leukocytes after surface stimulation.
2. Chemotaxin-induced changes in cyclic adenosine monophosphate levels in human neutrophils
3. Chemoattractant-elicited alterations of cAMP levels in human polymorphonuclear leukocytes require a Ca2+-dependent mechanism which is independent of transmembrane activation of adenylate cyclase.
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1. Montelukast inhibits neutrophil pro-inflammatory activity by a cyclic AMP-dependent mechanism;British Journal of Pharmacology;2009-01
2. A Chemoattractant-mediated Gi-coupled Pathway Activates Adenylyl Cyclase in Human Neutrophils;Molecular Biology of the Cell;2007-02
3. Taming the neutrophil: calcium clearance and influx mechanisms as novel targets for pharmacological control;Clinical and Experimental Immunology;2005-05-17
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5. Expression of adenylyl cyclase isoforms in neutrophils;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;2003-04
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