Assessing capreomycin resistance on tlyA deficient and point mutation (G695A) Mycobacterium tuberculosis strains using multi-omics analysis-Reference-Cited by-同舟云学术

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Assessing capreomycin resistance on tlyA deficient and point mutation (G695A) Mycobacterium tuberculosis strains using multi-omics analysis

Published:2019-11 Issue:7 Volume:309 Page:151323
ISSN:1438-4221
Container-title:International Journal of Medical Microbiology
language:en
Short-container-title:International Journal of Medical Microbiology

Author:

Zhao Jiao,Wei Wenjing,Yan Huimin,Zhou Ying,Li Zhenyan,Chen Yanmei,Zhang Chenchen,Zeng Jincheng,Chen Tao,Zhou Lin

Funder

National Infectious Disease Science and Technology Major Projects

National Natural Science Foundation of China

Science and Technology Planning Project of Guangzhou

Publisher

Elsevier BV

Subject

Infectious Diseases,Microbiology (medical),General Medicine,Microbiology

Reference62 articles.

1. Molecular basis for the selectivity of antituberculosis compounds capreomycin and viomycin;Akbergenov;Antimicrob. Agents Chemother.,2011

2. Phthiocerol dimycocerosates of M. Tuberculosis participate in macrophage invasion by inducing changes in the organization of plasma membrane lipids;Astarie-Dequeker;PLoS Pathog.,2009

3. Gene knockout reveals a novel gene cluster for the synthesis of a class of cell wall lipids unique to pathogenic mycobacteria;Azad;J. Biol. Chem.,1997

4. Targeted replacement of the mycocerosic acid synthase gene in Mycobacterium bovis BCG produces a mutant that lacks mycosides;Azad;Proc. Natl. Acad. Sci. U. S. A.,1996

5. inhA, a gene encoding a target for isoniazid and ethionamide in Mycobacterium tuberculosis;Banerjee;Science,1994

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