N-terminal eGFP-tagging of rabbit apolipoprotein A-I decreases expression and impairs cholesterol-efflux activity-Reference-Cited by-同舟云学术

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N-terminal eGFP-tagging of rabbit apolipoprotein A-I decreases expression and impairs cholesterol-efflux activity

Published:2023-05 Issue:5 Volume:1868 Page:159301
ISSN:1388-1981
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids

Author:

Wacker Bradley K.,Bi Lianxiang,Liu Li,Sorci-Thomas Mary G.,Ng Philip,Palmer Donna J.,Tang Chongren,Dichek David A.

Funder

National Institutes of Health

National Heart Lung and Blood Institute

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference16 articles.

1. High-density lipoprotein/apolipoprotein a-i infusion therapy;Tardif;Curr. Atheroscler. Rep.,2009

2. Csl112 (apolipoprotein a-i [human]) enhances cholesterol efflux similarly in healthy individuals and stable atherosclerotic disease patients;Gille;Arterioscler. Thromb. Vasc. Biol.,2018

3. Apolipoprotein a-i vascular gene therapy provides durable protection from atherosclerosis in hyperlipidemic rabbits;Wacker;Arterioscler. Thromb. Vasc. Biol.,2018

4. Hdl-mediated mechanisms of protection in cardiovascular disease;Kontush;Cardiovasc. Res.,2014

5. Deletion of the propeptide of apolipoprotein a-i reduces protein expression but stimulates effective conversion of prebeta-high density lipoprotein to alpha-high density lipoprotein;Sviridov;J. Lipid Res.,2000

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