Morphologic effects of sulfur-substituted fatty acids on rat hepatocytes with special reference to proliferation of peroxisomes and mitochondria
Author:
Publisher
Elsevier BV
Subject
Structural Biology
Reference29 articles.
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3. Correlation between the cellular level of long-chain acyl-coa, peroxisomal β-oxidation, and palmitoyl-CoA hydrolase activity in rat liver. Are the two enzyme systems regulated by a substrate-induced mechanism?
4. Alkylthioacetic acid (3-thia fatty acids) — a new group of non-β-oxidizable, peroxisome-inducing fatty acid analogues. I. A study on the structural requirements for proliferation of peroxisomes and mitochondria in rat liver
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1. Associations between fatty acid oxidation, hepatic mitochondrial function, and plasma acylcarnitine levels in mice;Nutrition & Metabolism;2018-01-29
2. The Roles of β-Oxidation and Cofactor Homeostasis in Peroxisome Distribution and Function in Arabidopsis thaliana;Genetics;2016-11-01
3. Regulation and Quantification of Cellular Mitochondrial Morphology and Content;Current Pharmaceutical Design;2014-09-12
4. An Immunomodulating Fatty Acid Analogue Targeting Mitochondria Exerts Anti-Atherosclerotic Effect beyond Plasma Cholesterol-Lowering Activity in apoE-/- Mice;PLoS ONE;2013-12-04
5. Induction of mitochondrial biogenesis and respiration is associated with mTOR regulation in hepatocytes of rats treated with the pan-PPAR activator tetradecylthioacetic acid (TTA);Biochemical and Biophysical Research Communications;2013-01
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