MEHP induces alteration of mitochondrial function and inhibition of steroid biosynthesis in MA-10 mouse tumor Leydig cells
Author:
Funder
University of Southern California
National Institutes of Health
North Carolina Biotechnology Center
Publisher
Elsevier BV
Subject
Toxicology
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1. Di-(2-ethylhexyl) phthalate exposure induces premature testicular senescence by disrupting mitochondrial respiratory chain through STAT5B-mitoSTAT3 in Leydig cell;GeroScience;2024-03-19
2. Prenatal DEHP exposure induces lifelong testicular toxicity by continuously interfering with steroidogenic gene expression;Translational Andrology and Urology;2024-03
3. Use of in vitro methodology to investigate phthalate effects on the differentiation of seminiferous tubule-associated stem cells to form Leydig cells and on the Leydig cells derived from the stem cells;Frontiers in Toxicology;2024-02-01
4. Prenatal DEHP Exposure Induces Premature Testicular Aging by Promoting Leydig Cell Senescence through the MAPK Signaling Pathways;Advanced Biology;2023-05-28
5. Urinary concentration of phthalates and bisphenol A during minipuberty is associated with reproductive hormone concentrations in infant boys;International Journal of Hygiene and Environmental Health;2023-05
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