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Increased endothelial nitric oxide production after low level lead exposure in rats involves activation of angiotensin II receptors and PI3K/Akt pathway

  • Published:2020-10 Issue: Volume:443 Page:152557
  • ISSN:0300-483X
  • Container-title:Toxicology
  • language:en
  • Short-container-title:Toxicology

Author:

Fiorim Jonaina,Simões Maylla Ronacher,de Azevedo Bruna Fernandes,Ribeiro Rogério Faustino,dos Santos LeonardoORCID,Padilha Alessandra SimãoORCID,Vassallo Dalton Valentim

Funder

Coordenação de Aperfeiçoamento de Pessoal de Nível Superior

Conselho Nacional de Desenvolvimento Científico e Tecnológico

Fundação Estadual de Amparo à Pesquisa do Estado do Espírito Santo

Publisher

Elsevier BV

Subject

Toxicology

Reference48 articles.

1. Toxicological Profile for Lead. Atlanta (GA): Agency for Toxic Substances and Disease Registry (US);Abadin,2007

2. Blood Pressure Decreases Following Lead Treatment Cessation: Highest NO Bioavailability Involved;Broseghini-Filho;Biol. Trace Elem. Res.,2016

3. Kininergic system and arterial hypertension following chronic exposure to inorganic lead;Carmignani;Immunopharmacology,1999

4. Catcholamine and nitric oxide systems as targets of chronic lead exposure in inducing selective functional impairment;Carmignani;Life Sci.,2000

5. Low-level lead exposure changes endothelial modulation in rat resistance pulmonary arteries;Covre;Vascul. Pharmacol.,2016

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