Prenatal drug exposures sensitize noradrenergic circuits to subsequent disruption by chlorpyrifos
Author:
Publisher
Elsevier BV
Subject
Toxicology
Reference75 articles.
1. Maternal exposure to nicotine and chlorpyrifos, alone and in combination, leads to persistently elevated expression of glial fibrillary acidic protein in the cerebellum of the offspring in late puberty;Abdel-Rahman;Arch. Toxicol.,2004
2. In utero exposure to nicotine and chlorpyrifos alone, and in combination produces persistent sensorimotor deficits and Purkinje neuron loss in the cerebellum of adult offspring rats;Abou-Donia;Arch. Toxicol.,2006
3. Developmental exposure of rats to chlorpyrifos leads to behavioral alterations in adulthood, involving serotonergic mechanisms and resembling animal models of depression;Aldridge;Environ. Health Perspect.,2005
4. Alterations in central nervous system serotonergic and dopaminergic synaptic activity in adulthood after prenatal or neonatal chlorpyrifos exposure;Aldridge;Environ. Health Perspect.,2005
5. Developmental exposure to chlorpyrifos elicits sex-selective alterations of serotonergic synaptic function in adulthood: critical periods and regional selectivity for effects on the serotonin transporter, receptor subtypes, and cell signaling;Aldridge;Environ. Health Perspect.,2004
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