Study on developmental abnormalities in hypospadiac male rats induced by maternal exposure to di-n-butyl phthalate (DBP)
Author:
Publisher
Elsevier BV
Subject
Toxicology
Reference43 articles.
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2. Pathogenesis of male reproductive tract lesions from gestation through adulthood following in utero exposure to Di(n-butyl) phthalate;Barlow;Toxicol. Pathol.,2003
3. Quantitative changes in gene expression in fetal rat testes following exposure to di(n-butyl) phthalate;Barlow;Toxicol. Sci.,2003
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1. Dibutyl phthalate (DBP) promotes Epithelial-Mesenchymal Transition (EMT) to aggravate liver fibrosis into cirrhosis and portal hypertension (PHT) via ROS/TGF-β1/Snail-1 signalling pathway in adult rats;Ecotoxicology and Environmental Safety;2024-04
2. Maternal exposure to dibutyl phthalate (DBP) impairs angiogenesis and AR signalling pathway through suppression of TGFB1I1 in hypospadias offspring;Ecotoxicology and Environmental Safety;2024-01
3. Effects of Dibutylphthalate and Steroid Hormone Mixture on Human Prostate Cells;International Journal of Molecular Sciences;2023-09-20
4. Di-n-butyl phthalate induces oversecretion of vascular endothelium-derived NAP-2 and promotes epithelial-mesenchymal transition of urothelial cells in newborn hypospadias rats;Ecotoxicology and Environmental Safety;2023-05
5. Identification of endocrine-disrupting chemicals targeting the genes and pathways of genital anomalies in males;Ecotoxicology and Environmental Safety;2022-12
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