Bmi1 signaling maintains the plasticity of airway epithelial progenitors in response to persistent silica exposures-Reference-Cited by-同舟云学术

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Bmi1 signaling maintains the plasticity of airway epithelial progenitors in response to persistent silica exposures

Published:2022-03 Issue: Volume:470 Page:153152
ISSN:0300-483X
Container-title:Toxicology
language:en
Short-container-title:Toxicology

Author:

Yang Jiali,Wu Shuang,Hu Wenfeng,Yang Dandan,Ma Jia,Cai Qian,Xue Jing,Chen Juan,Li Feng,Zeng Jing,Liu Xiaoming

Funder

Natural Science Foundation of Ningxia Province

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Toxicology

Reference41 articles.

1. Senescence induced by BMI1 inhibition is a therapeutic vulnerability in H3K27M-mutant DIPG;Balakrishnan;Cell Rep.,2020

2. Structure and E3-ligase activity of the ring-ring complex of polycomb proteins Bmi1 and Ring1b;Buchwald;EMBO J.,2006

3. Adenoviral transduction of dickkopf-1 alleviates silica-induced silicosis development in lungs of mice;Cai;Hum. Gene Ther.,2022

4. TGF-beta1/IL-11/MEK/ERK signaling mediates senescence-associated pulmonary fibrosis in a stress-induced premature senescence model of Bmi-1 deficiency;Chen;Exp. Mol. Med.,2020

5. Long-term exposure to silica dust and risk of total and cause-specific mortality in Chinese workers: a cohort study;Chen;PLoS Med.,2012

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2. Activation of Sirtuin3 by honokiol ameliorates alveolar epithelial cell senescence in experimental silicosis via the cGAS-STING pathway;Redox Biology;2024-08

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4. NLRP3 inflammasome mediates abnormal epithelial regeneration and distal lung remodeling in silica‑induced lung fibrosis;International Journal of Molecular Medicine;2024-01-16

5. Single-cell transcriptome sequencing–based analysis: probing the mechanisms of glycoprotein NMB regulation of epithelial cells involved in silicosis;Particle and Fibre Toxicology;2023-07-19

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