Mutation of the Human Circadian Clock Gene CRY1 in Familial Delayed Sleep Phase Disorder-Reference-Cited by-同舟云学术

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Mutation of the Human Circadian Clock Gene CRY1 in Familial Delayed Sleep Phase Disorder

Published:2017-04 Issue:2 Volume:169 Page:203-215.e13
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

Patke Alina,Murphy Patricia J.,Onat Onur Emre,Krieger Ana C.,Özçelik Tayfun,Campbell Scott S.,Young Michael W.

Funder

NIH

Weill Cornell Medical College

Calico Life Sciences LLC

The Rockefeller University Center for Clinical and Translational Science

the Turkish Academy of Sciences-TÜBA

The Rockefeller University Women & Science Postdoctoral Fellowship

Brain & Behavior Research Foundation

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference76 articles.

1. International Classification of Sleep Disorders: Diagnostic and Coding Manual,2005

2. Distinct and separable roles for endogenous CRY1 and CRY2 within the circadian molecular clockwork of the suprachiasmatic nucleus, as revealed by the Fbxl3(Afh) mutation;Anand;J. Neurosci.,2013

3. Hypersensitivity of melatonin suppression in response to light in patients with delayed sleep phase syndrome;Aoki;Chronobiol. Int.,2001

4. A length polymorphism in the circadian clock gene Per3 is linked to delayed sleep phase syndrome and extreme diurnal preference;Archer;Sleep,2003

5. Circadian behavior is light-reprogrammed by plastic DNA methylation;Azzi;Nat. Neurosci.,2014

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