Proapoptotic BID Is an ATM Effector in the DNA-Damage Response-Reference-Cited by-同舟云学术

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Proapoptotic BID Is an ATM Effector in the DNA-Damage Response

Published:2005-08 Issue:4 Volume:122 Page:593-603
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

Kamer Iris,Sarig Rachel,Zaltsman Yehudit,Niv Hagit,Oberkovitz Galia,Regev Limor,Haimovich Gal,Lerenthal Yaniv,Marcellus Richard C.,Gross Atan

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference18 articles.

1. Permanent cell cycle exit in G2 phase after DNA damage in normal human fibroblasts;Baus;EMBO J.,2003

2. Phosphorylation of bid by casein kinases I and II regulates its cleavage by caspase 8;Desagher;Mol. Cell,2001

3. DNA damage triggers a prolonged p53-dependent G1 arrest and long-term induction of Cip1 in normal human fibroblasts;Di Leonardo;Genes Dev.,1994

4. Dissection of a DNA-damage-induced transcriptional network using a combination of microarrays, RNA interference and computational promoter analysis;Elkon;Genome Biol.,2005

5. DNA damage checkpoint control in cells exposed to ionizing radiation;Iliakis;Oncogene,2003

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