A Surveillance Mechanism Ensures Repair of DNA Lesions during Zygotic Reprogramming-Reference-Cited by-同舟云学术

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A Surveillance Mechanism Ensures Repair of DNA Lesions during Zygotic Reprogramming

Published:2016-12 Issue:7 Volume:167 Page:1774-1787.e13
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

Ladstätter Sabrina,Tachibana-Konwalski Kikuë

Funder

VBC PhD programme in Molecular Life Sciences

FWF-funded DK Chromosome Dynamics

Austrian Academy of Sciences

European Research Council

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference70 articles.

1. De novo DNA methylation drives 5hmC accumulation in mouse zygotes;Amouroux;Nat. Cell Biol.,2016

2. Regulation of transcriptional activity during the first and second cell cycles in the preimplantation mouse embryo;Aoki;Dev. Biol.,1997

3. Breaching the DNA damage checkpoint via PF-00477736, a novel small-molecule inhibitor of checkpoint kinase 1;Blasina;Mol. Cancer Ther.,2008

4. Distinct but concerted roles of ATR, DNA-PK, and Chk1 in countering replication stress during S phase;Buisson;Mol. Cell,2015

5. Replication stress links structural and numerical cancer chromosomal instability;Burrell;Nature,2013

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