Tankyrase Loses Its Grip on SH3BP2 in Cherubism
Author:
Publisher
Elsevier BV
Subject
General Biochemistry, Genetics and Molecular Biology
Reference9 articles.
1. Identification of a Protein that Binds to the SH3 Region of Abl and Is Similar to Bcr and GAP-rho
2. Structural Basis and Sequence Rules for Substrate Recognition by Tankyrase Explain the Basis for Cherubism Disease
3. Tankyrase function at telomeres, spindle poles, and beyond
4. Loss of Tankyrase-Mediated Destruction of 3BP2 Is the Underlying Pathogenic Mechanism of Cherubism
5. Identification of a novel mutation ofSH3BP2in cherubism and demonstration thatSH3BP2mutations lead to increased NFAT activation
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1. Thoughts on the Etiology of Cherubism;Journal of Clinical Medicine;2024-04-03
2. Multiple versus solitary giant cell lesions of the jaw: Similar or distinct entities?;Bone;2021-08
3. Insights of tankyrases: A novel target for drug discovery;European Journal of Medicinal Chemistry;2020-12
4. Tankyrase (PARP5) Inhibition Induces Bone Loss through Accumulation of Its Substrate SH3BP2;Cells;2019-02-22
5. Autoinflammatory Bone Disorders;Textbook of Pediatric Rheumatology;2016
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